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Genetic Knockdown of Brain-Derived Neurotrophic Factor in 3xTg-AD Mice Does  Not Alter Aβ or Tau Pathology | PLOS ONE
Genetic Knockdown of Brain-Derived Neurotrophic Factor in 3xTg-AD Mice Does Not Alter Aβ or Tau Pathology | PLOS ONE

Systematic phenotyping and characterization of the 3xTg-AD mouse model of  Alzheimer's Disease | bioRxiv
Systematic phenotyping and characterization of the 3xTg-AD mouse model of Alzheimer's Disease | bioRxiv

Frontiers | Transcriptional, Behavioral and Biochemical Profiling in the  3xTg-AD Mouse Model Reveals a Specific Signature of Amyloid Deposition and  Functional Decline in Alzheimer's Disease
Frontiers | Transcriptional, Behavioral and Biochemical Profiling in the 3xTg-AD Mouse Model Reveals a Specific Signature of Amyloid Deposition and Functional Decline in Alzheimer's Disease

Fingolimod Rescues Memory and Improves Pathological Hallmarks in the 3xTg-AD  Model of Alzheimer's Disease | Molecular Neurobiology
Fingolimod Rescues Memory and Improves Pathological Hallmarks in the 3xTg-AD Model of Alzheimer's Disease | Molecular Neurobiology

IVIg protects the 3xTg-AD mouse model of Alzheimer's disease from memory  deficit and Aβ pathology | Journal of Neuroinflammation | Full Text
IVIg protects the 3xTg-AD mouse model of Alzheimer's disease from memory deficit and Aβ pathology | Journal of Neuroinflammation | Full Text

Dopaminergic dysfunction in the 3xTg-AD mice model of Alzheimer's disease |  Scientific Reports
Dopaminergic dysfunction in the 3xTg-AD mice model of Alzheimer's disease | Scientific Reports

IJMS | Free Full-Text | Whole Blood Transcriptome Characterization of 3xTg-AD  Mouse and Its Modulation by Transcranial Direct Current Stimulation (tDCS)
IJMS | Free Full-Text | Whole Blood Transcriptome Characterization of 3xTg-AD Mouse and Its Modulation by Transcranial Direct Current Stimulation (tDCS)

Frontiers | IntelliCage Automated Behavioral Phenotyping Reveals Behavior  Deficits in the 3xTg-AD Mouse Model of Alzheimer's Disease Associated With  Brain Weight
Frontiers | IntelliCage Automated Behavioral Phenotyping Reveals Behavior Deficits in the 3xTg-AD Mouse Model of Alzheimer's Disease Associated With Brain Weight

Peripheral adaptive immunity of the triple transgenic mouse model of  Alzheimer's disease | Journal of Neuroinflammation | Full Text
Peripheral adaptive immunity of the triple transgenic mouse model of Alzheimer's disease | Journal of Neuroinflammation | Full Text

IJMS | Free Full-Text | Both Amyloid-β Peptide and Tau Protein Are Affected  by an Anti-Amyloid-β Antibody Fragment in Elderly 3xTg-AD Mice
IJMS | Free Full-Text | Both Amyloid-β Peptide and Tau Protein Are Affected by an Anti-Amyloid-β Antibody Fragment in Elderly 3xTg-AD Mice

A History of Repeated Alcohol Intoxication Promotes Cognitive Impairment  and Gene Expression Signatures of Disease Progression in the 3xTg Mouse  Model of Alzheimer's Disease | eNeuro
A History of Repeated Alcohol Intoxication Promotes Cognitive Impairment and Gene Expression Signatures of Disease Progression in the 3xTg Mouse Model of Alzheimer's Disease | eNeuro

Pharmaceutics | Free Full-Text | Cognitive Impairment in the 3xTg-AD Mouse  Model of Alzheimer's Disease is Affected by Aβ-ImmunoTherapy and Cognitive  Stimulation
Pharmaceutics | Free Full-Text | Cognitive Impairment in the 3xTg-AD Mouse Model of Alzheimer's Disease is Affected by Aβ-ImmunoTherapy and Cognitive Stimulation

A model for IVIg mechanisms of action in the 3xTg-AD mice. Our... |  Download Scientific Diagram
A model for IVIg mechanisms of action in the 3xTg-AD mice. Our... | Download Scientific Diagram

Inhibition of HDAC3 reverses Alzheimer's disease-related pathologies in  vitro and in the 3xTg-AD mouse model | PNAS
Inhibition of HDAC3 reverses Alzheimer's disease-related pathologies in vitro and in the 3xTg-AD mouse model | PNAS

Suppressing aberrant phospholipase D1 signaling in 3xTg Alzheimer's disease mouse  model promotes synaptic resilience | Scientific Reports
Suppressing aberrant phospholipase D1 signaling in 3xTg Alzheimer's disease mouse model promotes synaptic resilience | Scientific Reports

miRNA-31 Improves Cognition and Abolishes Amyloid-β Pathology by Targeting  APP and BACE1 in an Animal Model of Alzheimer's Disease - ScienceDirect
miRNA-31 Improves Cognition and Abolishes Amyloid-β Pathology by Targeting APP and BACE1 in an Animal Model of Alzheimer's Disease - ScienceDirect

Frontiers | Systematic Phenotyping and Characterization of the 3xTg-AD  Mouse Model of Alzheimer's Disease
Frontiers | Systematic Phenotyping and Characterization of the 3xTg-AD Mouse Model of Alzheimer's Disease

Schematic representation of the study design. Six-month-old 3xTg-AD... |  Download Scientific Diagram
Schematic representation of the study design. Six-month-old 3xTg-AD... | Download Scientific Diagram

Expression of pathological factors in 3xTg-AD model mice following... |  Download Scientific Diagram
Expression of pathological factors in 3xTg-AD model mice following... | Download Scientific Diagram

Neurovascular uncoupling in the triple transgenic model of Alzheimer's  disease: Impaired cerebral blood flow response to neuronal-derived nitric  oxide signaling - ScienceDirect
Neurovascular uncoupling in the triple transgenic model of Alzheimer's disease: Impaired cerebral blood flow response to neuronal-derived nitric oxide signaling - ScienceDirect

The standardized Lycium chinense fruit extract protects against Alzheimer׳s  disease in 3xTg-AD mice - ScienceDirect
The standardized Lycium chinense fruit extract protects against Alzheimer׳s disease in 3xTg-AD mice - ScienceDirect

Morphological analysis of the hippocampal region associated with an innate  behaviour task in the transgenic mouse model (3xTg-AD) for Alzheimer  disease | Neurología (English Edition)
Morphological analysis of the hippocampal region associated with an innate behaviour task in the transgenic mouse model (3xTg-AD) for Alzheimer disease | Neurología (English Edition)

Tetracyclic Truncated Analogue of the Marine Toxin Gambierol Modifies NMDA,  Tau, and Amyloid β Expression in Mice Brains: Implications in AD Pathology  | ACS Chemical Neuroscience
Tetracyclic Truncated Analogue of the Marine Toxin Gambierol Modifies NMDA, Tau, and Amyloid β Expression in Mice Brains: Implications in AD Pathology | ACS Chemical Neuroscience

PAK Inactivation Impairs Social Recognition in 3xTg-AD Mice without  Increasing Brain Deposition of Tau and Aβ | Journal of Neuroscience
PAK Inactivation Impairs Social Recognition in 3xTg-AD Mice without Increasing Brain Deposition of Tau and Aβ | Journal of Neuroscience